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However, H 2 S is an endogenous gasotransmitter, and accurately controlling its effective concentration in vivo is challenging

BDG induced increased phagocytic capacity of human monocytes in a GSH-dependent manner ( Figure 7C )

We found that overexpression of GmMORF2a or SsCm1 in DD background ( DD/GmMORF2a or DD/SsCm1 ) inhibited cell death and ion leakage after dexamethasone treatment, and inhibited ROS accumulation prior to visible cell death (Fig

The synthesis of GSH occurs by the consecutive involvement of enzymes -glutamylcysteine synthetase and glutathione synthetase [2]

Glucose influence on germ tube production in Candida albicans