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10.1016/j.bbih.2023.100627 446 UngerM

On the basis of these studies, the capacity of HGF to modulate neuro-inflammation is thus associated with an alteration in the balance of pro-inflammatory (Th1 and Th17) and anti-inflammatory (Th2 and Tregs) CD4 + T cells by influencing dendritic cell function

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However, the experimental data exhibits and offset of 6% corresponding to an analgesic effect in the absence of drug administration that cannot be explained by the paradigm of an increase in anionic current