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The difference between the effects of both QIs on the cell viability ( Figure 2 ) and caspase 3 activation ( Figure 4 ) suggests that upon GSH depletion, the decrease in cell viability is not only apoptosis dependent but also most likely depends other cell death pathways, such as necrosis (Iorga et al., 2017)

3c, d) upon siRNA-mediated knock-down of the GR1 (Fig

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Theoretical basis, experimental design, and computerized simulation of synergism and antagonism in drug combination studies