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for example, the autophagy receptor hippocalcin-like 1 (HPCAL1) promotes ferroptosis yet paradoxically facilitates NSCLC growth in vivo, suggesting that its net oncogenic role may involve pleiotropic functions beyond regulating this cell death pathway (77)

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Current experimental datasets from animal studies do not show tumor-promoting effects in laboratory conditions, though theoretical considerations regarding angiogenesis pathway modulation exist in research literature

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