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Cathepsin C is a tissue-specific regulator of squamous carcinogenesis

This duality positions GSH as a context- and compartment-dependent determinant of cellular redox thresholds and raises several unresolved questions that will shape future directions in redox biology and cell death research

In the pancreas, amylin suppresses post-prandial glucagon release

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Establishment by pathogenic bacteria triggers the secretion of various abnormal metabolites, such as sialidase, -glucuronidase, coagulase, and neuraminidase, which diminish the thickness of the cervicovaginal fluid (CVF) and impair its protective role