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Cigarette smoking promotes acid reflux through multiple mechanisms including reduced lower esophageal sphincter pressure, increased transient sphincter relaxations, impaired esophageal clearance, and decreased saliva production

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Conclusion The recognition of the molecular mechanisms of the epigenetic changes in abnormal inflammation is expected to contribute to the understanding of the pathophysiology of CS-related diseases such that novel epigenetic therapies may be identified in the near future

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