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Previous studies indicated that lysosomal iron is released and translocated into the mitochondria contributing to MPT in APAP-induced injury ( 2+ uniporter MCFU plays a key role in APAP-induced oxidative stress, mitochondrial dysfunction, and hepatocellular death ( 2+ from lysosomes followed by uptake into mitochondria via MCFU is a key mechanism in APAP-mediated hepatotoxicity

10.1093/hmg/ddp326 Hum

Figure 2 illustrates the dosing schedule and blood sampling scheme for the pharmacokinetic phase of the study

"ALCAR for prevention of taxane-induced neuropathy." J Clin Oncol 2018;36(20):2048-55