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[DOI] [Google Scholar] Pastore A., Federici G., Bertini E., Piemonte F
GHK occurs endogenously in human plasma, with reported levels declining markedly with age
Ongoing waves of SARS-CoV-2 infection sustain the scenario where in the absence of improved vaccines or immunotherapies, clinically vulnerable individuals will still require annual or biannual vaccinations with updated immunogens matched to circulating variants
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Increased Production of Bilirubin Fetalmaternal blood group incompatibilities Extravascular blood in body tissues Polycythemia Red blood cell abnormalities (hemoglobinopathies, membrane and enzyme defects) Induction of labor Decreased Excretion of Bilirubin Increased enterohepatic circulation of bilirubin Breast-feeding Inborn errors of metabolism Hormones and drugs Prematurity Hepatic hypoperfusion Cholestatic syndromes Obstruction of the biliary tree Combined Increased Production and Decreased Excretion of Bilirubin Sepsis Intrauterine infection Congenital cirrhosis The high incidence of jaundice in otherwise completely normal neonates has resulted in the term physiologic jaundice
