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and (2) the serum cotinine level does signify the accumulated SHS exposures, but it will not increase linearly with the exposure time for long-term inhalation of tobacco smoke due to continuous metabolism of cotinine

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This implies that the impact of CSE on HIF-1 is more evident under normoxic conditions, as the significant stabilization of HIF-1 in hypoxic conditions (due to O 2 -dependent inhibition of PHDs) could obscure the effect of CSE

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