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[1] At the cellular level, the tendon work is genuinely mechanistic: in cultured rat tendon fibroblasts, BPC-157 accelerated cell outgrowth, improved survival under oxidative stress, and increased migration in a dose-dependent way, apparently through the FAKpaxillin pathway

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The vasoprotective effects align neatly with the mechanistic picture: a compound that promotes angiogenesis and modulates nitric oxide would predictably stabilize vascular function across a range of stressors