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Under pathological conditions, FUNDC1 persists in a phosphorylated state that specifically impedes its molecular interaction with the autophagosomal marker protein LC3-II, consequently suppressing mitophagy initiation and ultimately inducing neuronal dysfunction ( Mitochondrial fragmentation has also been demonstrated in AD patients and model animals

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As of December 2023, out of the 250 current active pharmacological and non-pharmacological NIH clinical trials there is no specific mention of trials stated to target microbes (National Institute on Aging, 2024)

A similar finding was also observed in another small pilot study that compared subcutaneous vs