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3.5.2 GPX4-regulated neutrophil ferroptosis triggers autoimmunity Neutrophilscentral effectors of innate immunityare now implicated in the pathogenesis of SLE not only through classical functions (e.g., autoantibody formation and TNF- release) but also via dysregulated iron metabolism and ferroptosis

proposed that commensal microbiota may be an important environmental factor in triggering HT [56]