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PLoS ONE 16(3):e0249029

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Beyond promoting autophagic flux to clear dysfunctional mitochondria, AMPK also reduces reactive oxygen species (ROS)-mediated activation of the NOD-like receptor protein 3 (NLRP3) inflammasome, subsequently suppressing the release of proinflammatory cytokines such as interleukin-1 (IL-1) and interleukin-18 (IL-18)

Research use differs from medical use because a laboratory or preclinical study does not establish an approved human treatment