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As predicted, the specific GST inhibitors displayed parasite growth inhibition at EC 50 of 6.9 and 4.4 M for S-hexylglutathione and ellagic acid, respectively (Supplementary Figure S2C)

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Our results demonstrated that TG2 mediates GST tag crosslinking or polyamination and causes aggregate formation

This activation leads to decreased plasma autoantibody levels, reduced inflammatory cytokine expression, and an overall improvement in LN pathology [158]

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