Targeting CD38 improves metabolic fitness and memory in 5xFAD mice via modulation of pathogenic T H 17-mediated immunity To address the potential mechanistic relationship between CD38 and altered T cell immunometabolic functions, we used the 5xFAD transgenic mouse model of AD
Takahashi, C., Muramatsu, R., Fujimura, H., Mochizuki, H
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Platelets triggered by thrombin had decreased levels of vWF binding sites on the GPIb-IX complex but increased levels of GPIV critical to thrombospondin, shown in Figure 5 (146)