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Description

According to the static PDB structure, it is difficult for inhibitors to interact with copper ions and inhibit tyrosinase activity (Fig

Neuroprotective effects of preconditioning ischemia on ischemic brain injury through down-regulating activation of JNK1/2 via N-methyl-D-aspartate receptor-mediated Akt1 activation

J Anc Dis Prev Rem.2014;2:2:1000111

And for longevity medicine, it validates something bigger: that one of the core hallmarks of aging, mitochondrial dysfunction, is now a druggable target