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339 Additionally, PFKFB3-driven glycolysis also promotes histone Kla and boosts profibrotic gene expression in silica-treated mice, a process that can be inhibited by activating glucagon-like peptide-1 receptor (GLP-1R), disrupting the interaction between the NLRP3 inflammasome and PFKFB3, and intensifying pulmonary fibrosis progression

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[DOI] [PubMed] [Google Scholar] Kraus T., Uttamsingh V., Anders M.W., Wolf S

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