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Inability to produce NADPH due to glucose 6-phosphate dehydrogenase (G6PD) deficiency renders an individual prone to infections, presumably due to reduced activity of NOX2, and susceptible to oxidative stress due to reduced activity of glutathione peroxidases (Luzzatto et al

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The journey began in the 1980s with research on Gila monster venom, culminating in the discovery of exendin-4, a stable, effective peptide for treating diabetes, demonstrating the progression from initial discovery to blockbuster diabetes medications

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The ideal cytoprotective agent should be effective both before and after injury induction, exerting continuous innate cell protection