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Description

The simultaneous interaction with the endogenous Ub-E2-E3 complex and the protein of interest results in the generation of an Ub-E2-E3-neosubstrate complex, ultimately leading to ubiquitinylation and degradation of target protein

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Its presence in bile was further validated by reanalysis of bile 16S rRNA datasets (Supplementary Tables S2, S3)

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Those are different content types

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Likewise, skin defect models (Rantfors and Cassuto, 2003

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