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Astrocytes can re-enter the cell cycle and proliferate in response to overt tissue damage such as is caused by stroke, severe trauma, infection, foreign bodies (including medical implants), autoimmune inflammation, neoplasm or severe neurodegeneration

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Concurrent impairment of nucleus and mitochondria for synergistic inhibition of cancer metastasis

The distinct metabolic preferences of Th17 (glycolysis and FAS) and Treg cells (OXPHOS and FAO), underscore the potential of metabolic reprogramming as a therapeutic strategy

The sustained oxidative stress status in KO livers was paralleled by increased levels of transcription factor NRF2 in the nucleus (Fig