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Additional mechanisms linking oxidative stress and sleep include the accumulation of extracellular adenosine, resulting from inhibition of adenosine kinase, that activates adenosine A1 receptors to promote sleep (Park and Gupta, 2013

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Additionally, -sitosterol inserted into the lipid bilayer of PEG-LNPs induces the formation of polyhedral particle shapes, which increase membrane contact area and promote membrane destabilization, thus enhancing endosomal escape after cellular uptake 172