Prevents dryness and scaling With low supply of vitamin B12, your skin barrier becomes thinner and weaker, which means it cant retain moisture effectively
(M+D)/2 upregulated TTC5 and HMGCR, involved in p53-dependent apoptosis and cholesterol metabolic process, respectively ( Supplementary Table S1 )
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Another proposed mechanism involves competing interactions at growth factor receptor sites
Measures of general nutritional status indicated that the association of increased homocysteine levels and diminished vitamin B 12 status with Alzheimer's disease was not due to dementia-related malnutrition (70)