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The accumulation of toxic lipids may cause sustained activation of JNK via ROS and MAP3K activation, while inhibition of JNK attenuates FFA-induced lipoapoptosis [69]

Joachimiak, Carla B

10.1371/journal.pone.0108210 41 TirmensteinM

In physiological conditions, selenoproteins are an effective defense system against a large amount of ROS, which thyrocytes constantly generate on their surface in order to accept electrons from oxidative reactions (59)