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So I am writing this in two parts

It has been shown that pathogenic bacteria or viruses in inflamed tissues promote ferroptosis in cells while activating immune cells and triggering inflammatory responses through the action of lipopolysaccharides, peptidoglycans, bacterial or viral DNA, and host cell toll-like receptors carried or released by themselves ( 3.2 Ferroptosis and IBD in the intestinal epithelium The intestinal epithelium is the first line of defense for the intestinal mucosa

They can be used to induce immune tolerance or to modulate the immune response in autoimmune diseases

Semax Peptide Reduces Lipopolysaccharide-Induced Systemic Inflammation and Intestinal Permeability in Mice

The results are remarkable