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Tryptophan metabolites in fibrosis regulation Tryptophan metabolism plays a complex, context-dependent role in pulmonary fibrosis, influencing ECM deposition, fibroblast activation, and immune responses

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On the contrary, the transcriptional repression of GAP1 was strong when the NADP-GDH activity was restored by expressing the GDH3 gene, and partially restored, when the bacterial GDHA was expressed in gdh1 mutant strains [34]