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Multiple oestradiol functions inhibit ferroptosis and acute kidney injury

Many studies examine isolated mechanisms rather than long-term patient outcomes

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When vitamin C deficiency or metabolic disorders are suspected

This review integrates mechanistic insights with translational advances, outlining how dysregulation of chaperones, autophagy-mitophagy, the ubiquitinproteasome system, and ER stress pathways drive human diseases, while highlighting emerging therapeutic platforms, from pharmacological chaperones and autophagy modulators to targeted protein degradation technologies, CRISPR screens, spatial biology, and AI-guided drug discovery