WES and/or WGS studies had not distinguished between smoking and non-smoking individuals until 2021, when results emerged from the two formerly discussed somatic alteration profiling studies of never-smoking LUAD, which included data from matched non-malignant tissue analyses ,7,78
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It has been proposed that ATXN3 is also degraded through autophagy [83], being the mutant (soluble) protein more prone to autophagic degradation, as no changes in endogenous wild-type ATXN3 levels were observed in drug-induced autophagy activation in vivo [84]
