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The causal role of OS in BPH has been supported by Vital et al.s (210) study, in which they generated a transgenic mouse model with overexpression of Nox4 under the control of the prostate-specific promoter ARR2PB

At 2 h post-APAP treatment, hepsin / mice also exhibited a greater reduction in total GSH compared with wild-type mice (Fig

Subgroup analyses and meta-regressions were performed on homocysteine, methionine, cysteine, vitamin B9, vitamin B12, vitamin D, and GSH because these markers were measured in more than ten studies, and the levels of these markers were dysregulated in children with ASD

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