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Elevated expression of AARS1 and lactylation of p53 are often associated with poor prognosis of patients, and administration of -alanine to antagonize the lactylation of p53, coupled with doxorubicin to activate the transcriptional activity of p53, can significantly inhibit the proliferation of tumor cells in animal models [23]

Concurrently, GSHa critical intracellular antioxidantbecomes depleted, compromising the cell's capacity to neutralize ROS and lipid peroxidation products, thereby accelerating ferroptotic progression (21)

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however, only 12% of those patients presenting to this same center with acute liver failure from APAP overdose actually received a transplant