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The vasodilatory effects of NO also facilitate greater oxygen delivery, promote leukocyte infiltration, and enhance the microbicidal activity of macrophages [15,16]

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Recently, it was shown that, in the skeletal muscle of cachectic patients, there is increased expression and activity of TNF--related pathways, including TNF- mRNA, activation of TNFR1, and TNF- associated to TNFR1, and increased oxidative stress [41]

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However, their dysregulation can exacerbate inflammation and tissue damage in ARDS ( Given the central role of oxidative stress and lipid peroxidation in ARDS, targeting ferroptosis represents a promising therapeutic strategy ( 2 Ferroptosis in infection and ARDS development Cell death pathways play critical roles in immune homeostasis, infection defense, and inflammation resolution