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In resolved mice, UPEC initially form intracellular bacterial communities in the superficial facet cells, similar to that in adult nave mice, but they are rapidly shed within the first 6 h of infection via enhanced TNF-mediated inflammation 7

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NTZ replicated findings similar to those of the ER stress inhibitor (4-PBA) used in the study, further supporting our conclusion

This approach recognizes that inflammation serves as both an initiator and perpetuator of autoimmune processes, making inflammatory reduction a cornerstone of effective treatment

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