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The mechanism involves inhibition of nitric oxide synthase (NOS) activity: glutathione binds nitric oxide (NO), a critical neurotransmitter in gut motility, effectively sequestering it before it can signal smooth muscle relaxation and coordinated peristaltic waves

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Some have shown promising therapeutic effects in preclinical models of disease

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These further suggested that supplementation of GSH or Fer-1 mitigated meiotic inhibition and acrosomal dysplasia owing to repress testis ferroptosis, further alleviating oligospermia and teratospermia in older mice