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Induction of apoptosis in human breast cancer by adenoviral mediated overexpression of the proapoptotic gene bax

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The abnormal activation and inflammatory response of astrocytes may release some harmful substances, such as ROS, to further damage the surrounding neurons (Emerit et al., 2004

Oxidative stress arises from an imbalance between the production of reactive oxygen species (ROS) and the body's antioxidant defense mechanisms

Previous research suggested that TERT might contribute to lifespan extension through oxidative stress modulation and protection from oxidative damage, which is known to contribute to aging [5]