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Inflammatory cytokines secreted in the inflammatory area, such as granulocyte-macrophage colony stimulating factor (GM-CSF), TNF-, IL-1, and interferon, can activate HIF-1 to inhibit neutrophil apoptosis (62, 63)

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Similar content being viewed by others Introduction Glucagon-like peptide-1 receptor agonists (GLP-1 RAs) are structurally modified analogs of endogenous GLP-1, engineered to resist enzymatic degradation while retaining potent agonism at the GLP-1 receptor (GLP-1R) 1

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