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indeed, with respect to malaria protection, it has been theorized that protection conferred by G6PD deficiency may be related to increased phagocytosis of parasitized oxidatively-damaged G6PD-deficient RBCs (7779) via binding by macrophage Fc and lectin-like receptors (80), and that such parasites may be more susceptible to ROS (81)

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Given the drawbacks of drug-based antioxidative therapies, diet-based approaches present an alternative approach to protecting against OS

Within those categories, the clinical picture is clearer than the internet makes it seem