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10.1016/j.celrep.2014.03.048 15 ChiZ.ZhangJ.TokunagaA.HarrazM

Following chronic liver injury, HSCs proliferate, lose their vitamin A and undergo a major phenotypical transformation to -smooth muscle actin (-SMA) positive activated HSCs, which produce a wide variety of collagenous and non-collagenous extracellular matrix (ECM) proteins [168]