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NNMT has been shown to be present in the human brain, a necessity for neurotoxicity, because charged compounds cannot cross the bloodbrain barrier (Williams & Ramsden, 2005)

These changes markedly reduce its affinity for the insulin-like growth factor-binding proteins (IGFBPs) while retaining agonist activity at the IGF-1 receptor, characteristics that make it a useful reference ligand in growth-factor biology

Inadequate deep sleep may impair this clearance, potentially contributing to neurological disorders

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