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Although the interaction between endothelial dysfunction and vascular complications of diabetes is not fully understood, the existing view shows that a high glucose environment leads to the impairment of endothelium-dependent vasodilation capacity by increasing oxidative stress and inflammation, reducing vasodilation factor activity, increasing reactive oxygen species (ROS) production, promoting the uncoupling of endothelial nitric oxide synthase (eNOS), and so on ( Figure 2 )
Bae, T., Hallis, S
The tissues in your eyes are particularly sensitive to oxidative damage because they are constantly exposed to light and oxygen
In patients with COPD, activation and degranulation of neutrophils result in the release of activated NSPs, primarily NE, PR3, and CatG (54), disrupting the normal balance between proteases and antiproteases (53)
ALOX12, for example, promotes TP53-dependent iron apoptosis in specific cancer cells (61)
