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Caspase-independent pathways of hair cell death induced by kanamycin in vivo

As consequence, NRF2 overexpression increases the expression of SLC7A11 as well as other antioxidant target genes, resulting in enhanced GSH production (19)

No associations of fibrinolysis inhibitors changes with PC on treatment were found in either patient group

Without adequate VEGF signaling, wound healing stalls: fibroblasts deposit matrix without vascular support, creating avascular scar tissue rather than functional regeneration

Biotin-labelled cDNA was hybridised to MouseWG-6 expression bead chips, and scanning was performed using an Illumina BeadArray Reader