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Hepatic effects that are considered adverse include death, abnormal clinical chemistry parameters, obvious functional organ impairment, DNA and mitochondrial alteration, glutathione depletion, hepatocyte necrosis, enhanced replicative DNA synthesis, generation of reactive oxygen species, and increased peroxisome proliferation (in rodents) (Williams and Iatropoulos 2002)

Fabrication of Nanoparticles based on Hesperidin-Loaded Chitosan- Functionalized Fe 3 O 4 : Evaluation of In vitro Antioxidant and Anticancer Properties

Researchers use Glutathione when examining antioxidant peptide chemistry, redox pathway behavior, and tripeptide stability under various controlled research conditions

There are two parts to this: the intrinsic low solubility of niclosamide in aqueous solution compared to its efficacy

Its mechanism of action exhibits high substrate specificity