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Therefore, the pro-apoptotic effect of tamoxifen is partially dependent on the inhibition of FLIP expression and inhibition of pAkt and stimulation of caspase activity in vivo and in vitro, respectively [87]

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NO overrelease in vitro) (Sikiric et al., 1997)

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Neutrophil extracellular traps exacerbate ischemic brain damage

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Bleeding gums or mouth ulcers

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It is metabolized into several peptide fragments, primarily involving proline-rich sequences, and eliminated with a half-life under 30 minutes

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