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Ligand binding triggers autophosphorylation of tyrosine residues in the kinase domain, recruiting IRS-1 and activating the PI3K/Akt/mTOR axis

TB-500 complements this process by regulating actin polymerization, a critical mechanism for cell motility, differentiation, and tissue remodeling

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MRI-guided platforms support noninvasive assessment of tumor distribution, vascular permeability and response, while PET- and SPECT-based approaches provide quantitative whole-body biodistribution data for tumor accumulation, off-target exposure and patient-level variability [72, 74, 86,87,88]

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