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The mechanism may be to prevent oxidative stress damage, and tyrosinase is effectively exported from endoplasmic reticulum (Guan et al., 2015)

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[65,66] hCDR1 treatment delayed the production of both anti-DNA antibodies and proteinuria, and increased the survival of lupus-prone mice by regulating B cell activating factor (BAFF)

However, contrasting NE infusion rate at baseline, we did not observe significant between-group differences in NEE dynamics pre-AVP initiation as was previously proposed [11]