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Therefore, the use of either miRNA antagonists, agonists, or miRNA mimics that inhibit c-Met signaling is a potential and promising treatment for cancer [165]

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There were faster granulation tissue formation, re-epithelialization, dermal remodeling, and collagen deposition through extracellular signalregulated kinases (ERK)1/2 signaling pathway as well as its downstream targets, including c-Fos, c-Jun, and egr-1