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Dysbiosis amplifies barrier dysfunction and immune activation, while inflammation further perturbs microbial composition and impairs epithelial repair, establishing a self-sustaining pathological cycle

In summary, the metabolic programs governing Th17 and Treg cell differentiationglucose utilization, lipid metabolism, and amino acid processingoperate as an integrated network rather than isolated pathways, dynamically interacting through central checkpoint nodes such as the mTOR-AMPK-HIF-1 axis

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Molecular docking software UCSF DOCK 6.5 (University of California) was used to test molecular docking simulations of PQQ with SOD1 and SOD2 protein, respectively

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