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Glutathione deficiency during early postnatal development caused schizophrenia-like symptoms and a reduction in BDNF levels in the cortex and hippocampus of adult Sprague-Dawley rats

Laboratory studies investigate how senescent microglia and astrocytes contribute to neuroinflammation and disease progression in neurodegenerative disease models, while FOXO4-DRI provides researchers with tools for testing whether senescent cell elimination can restore brain function and reduce neuroinflammatory markers

Similar to nonsilent immune forms of regulated necrosis, ferroptotic cell death can release damage-associated molecular patterns (DAMPs) that promote the development of multiple inflammatory diseases and trigger the innate immune system

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