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Upon induction of hypoxia, HIF1 is no longer polyubiquitinated and can form a functional heterodimer with HIF1, allowing it to translocate to the nucleus and induce transcription (261)

Despite its peptide nature, which often poses bioavailability challenges, Epitalon has been reported in some sources to exhibit resistance to hydrolysis, suggesting potential for oral uptake under specific conditions [1,4, 10]

The proposed chain of effects looks like this: NNMT inhibited nicotinamide is no longer inactivated by methylation and becomes available for conversion to NAD+

So just to orient you, this is the eye on the right

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