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In the central nervous system, glia-derived EVs support neuronal homeostasis under physiological conditions but can become pathogenic during HIV-1 infection and neuroinflammation by delivering inflammatory signals, stress-response proteins, viral small RNAs, HIV proteins, and other neurotoxic mediators that impair neuronal function [54,55,56]

Infiltration of CD8+ T-cells has been detected in epileptogenic areas, and it is considered that the greater infiltration of CD8+ T-cells in the CA1 region of the hippocampus shows a greater positive correlation with neuronal loss in the area (reviewed in [59,60]) (Figure 4)

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Dihydrolipoic acid may also reduce the oxidized form of -tocopherol (vitamin E) directly or indirectly through regenerating oxidized vitamin C (see the article on Vitamin E) (18) or oxidized coenzyme Q 10 (see the article on Coenzyme Q 10 ) (19)