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IRF3 enhances the interaction between YAP and TEAD4 in the nucleus, promoting nuclear translocation and activation of YAP (Jiao et al., 2018)

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In glutamine-addicted squamous cell carcinoma (SCC) cancer cells, V-9302 treatment inhibits the YAP1/TEAD signaling pathway to weaken the SCC cancer phenotype (Mickle, 2022)

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Batch: GF-AHK100-B242 Key analytical results include: Assay Content: 124.6 mg Identification Retention Time: 0.991 Identification Spectrum: 1000 Purity: greater than 99.8 percent Molecular Ion Mass Identification (MS Deconvolution): confirmed Bacterial Endotoxins: below 0.001 EU per mg Total Aerobic Microbial Count: not detected Total Yeast and Mold Count: not detected Product Overview AHK-Cu is a copper-binding synthetic peptide complex studied in controlled laboratory environments focused on peptide-mediated signalling pathways and molecular regulatory systems

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