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With varying half-lives among its components, regular dosing is common in research cycles

A summary of these key results is found in Fig

It has been suggested that the complete loss of uricase activity occurred gradually as a result of three successive mutations in the uricase genea nonsense mutation in codon 33 in exon 2, a nonsense mutation in codon 187 and a splice mutation in exon 3 (,)

Dynamic data was first fit into kinetic models with T 1 as an unknown parameter to estimate apparent in vivo T 1 of VitC, which was subsequently incorporated into the kinetic model as a fixed parameter to generate k assuming T 1 is the same across the subjects

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